She did not have FVE, so I am wondering why her H&H, and RBC's were low, and not her platelets? Would it have something to do with the production of erythpoiten impaired with the kidney? This one has stumped me, she did not have any other disease processes going, no chronice renal failure, dialysis, etc. She was malnourished, and on TPN regular lipids at 39 ml/hr with free water, via triple lumen subclavian. WBC:15.9, neutropjils 90 %, (although only trace bacteria was noted in her UA, white cells in UA was 5-10) She was receiving Levaquin and Vanc IV This conclusion is based on the following data: (1) circulating TPO is significantly decreased in GPIb-deficient Bernard-Soulier syndrome patients and in Gp1b / mice, compared with controls (see figure) (2) lower TPO levels in GPIb-deficient conditions were not attributable to increased TPO clearance by platelets lacking GPIb, but. ureter) She presented with hematuria, in her UA it just stated "too numerous" under RBC's present maintained over 30ml/hl of output, due to having a stent placed in the rt. She has hydronephrosis, and is schduled for surgery to remove pelvic tumors that have caused partial urinary obsturction on rt. Pt is 89 y/o Asian female, 85 pounds, no hx of anemia, only hx of HTN controlled with Toprol. On our careplans, we attempt to figure out why there are any abnormalities, and I stumped on this one.any input is appreciated:
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